Diathesis–stress model
Diathesis–Stress Model is a psychological theory that attempts to explain the cause of certain disorders, such as mental health disorders, based on the interaction of genetic and environmental factors. The term "diathesis" refers to a person's genetic vulnerability or predisposition to developing a disorder, while "stress" refers to environmental stressors or life events that may trigger the onset of the disorder.
Overview[edit | edit source]
The Diathesis–stress model posits that individuals inherit certain vulnerabilities that make them more or less susceptible to developing psychological disorders under certain environmental conditions. This model is used in many fields of psychology, including clinical psychology, health psychology, and abnormal psychology.
Diathesis[edit | edit source]
In the context of the diathesis-stress model, a diathesis is not a disorder itself, but rather a predisposition towards developing a disorder. This predisposition can be biological, such as a genetic tendency towards a certain condition, or psychological, such as a personality trait that makes an individual more susceptible to stress. Diatheses are thought to interact with stressors to trigger the onset of a disorder.
Stress[edit | edit source]
Stress, in the diathesis-stress model, refers to life events or environmental conditions that can trigger the onset of a disorder in an individual with a diathesis. These stressors can be a wide range of events or conditions, from major life events like the loss of a loved one, to daily hassles like traffic or work stress.
Applications[edit | edit source]
The diathesis-stress model has been applied to a wide range of psychological disorders, including depression, anxiety disorders, schizophrenia, and eating disorders. It has also been used to understand the development of physical health conditions, such as heart disease and cancer.
Criticisms[edit | edit source]
While the diathesis-stress model has been influential in our understanding of the development of psychological disorders, it has also been criticized for its simplicity. Some critics argue that the model does not take into account the complex interplay of genetic and environmental factors that contribute to the development of disorders.
See also[edit | edit source]
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Contributors: Prab R. Tumpati, MD