Tabun

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Tabun is an organophosphate nerve agent. It is colorless, tasteless, and odorless, and is lethal even at very low concentrations. Discovered in 1936 by German chemist Gerhard Schrader, it was originally developed as a pesticide before being used as a chemical weapon during World War II.

History[edit | edit source]

Tabun was the first of the so-called "G-series" nerve agents to be discovered. Schrader was working for the German company IG Farben when he made the discovery. He was attempting to develop a more effective pesticide, but instead created a substance that was incredibly toxic to humans. The German military quickly recognized the potential of this new chemical, and by 1939, they had begun mass production.

Properties and Effects[edit | edit source]

Tabun is a liquid at room temperature, but it can easily evaporate into a gas. It is less volatile than some other nerve gases, which means it persists in the environment for longer. Exposure to tabun can occur through inhalation, ingestion, or skin contact.

The effects of tabun are rapid and severe. It inhibits the enzyme acetylcholinesterase, which is necessary for the proper functioning of the nervous system. This leads to a buildup of acetylcholine in the body, causing a range of symptoms including convulsions, paralysis, respiratory failure, and death.

Treatment and Antidotes[edit | edit source]

Immediate treatment for tabun exposure is critical. The antidote, pralidoxime, must be administered as soon as possible after exposure. This drug reactivates acetylcholinesterase and helps to restore normal nervous system function. Other treatments include removing contaminated clothing, washing the exposed skin, and providing supportive care.

See Also[edit | edit source]


Tabun Resources

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