Ventricle (heart)
(Redirected from Ventricular pressure)
Ventricle (Heart)[edit | edit source]
A ventricle is a hollow chamber found in the heart, responsible for pumping blood to various parts of the body. In humans and most mammals, there are four chambers in the heart, with two ventricles located at the bottom.
Anatomy[edit | edit source]
The heart is divided into two sides, the left and the right, each containing an atrium and a ventricle. The left ventricle is responsible for pumping oxygenated blood to the rest of the body, while the right ventricle pumps deoxygenated blood to the lungs for oxygenation.
The walls of the ventricles are thicker and more muscular compared to the atria. This is because the ventricles need to generate enough force to propel blood throughout the body. The left ventricle, in particular, has the thickest walls due to its role in systemic circulation.
Function[edit | edit source]
The ventricles play a crucial role in the circulatory system. When the heart contracts, the ventricles contract simultaneously, pushing blood out of the heart and into the arteries. This process is known as systole. The left ventricle contracts more forcefully than the right ventricle, as it needs to pump blood to the entire body.
During diastole, the ventricles relax and fill with blood from the atria. The atrioventricular valves, such as the mitral valve on the left side and the tricuspid valve on the right side, prevent backflow of blood into the atria during ventricular contraction.
Clinical Significance[edit | edit source]
Various conditions can affect the ventricles and their function. One common condition is ventricular hypertrophy, which refers to the thickening of the ventricular walls. This can be caused by high blood pressure or heart diseases, and it can lead to reduced ventricular function.
Another condition is ventricular arrhythmia, which is an abnormal heart rhythm originating from the ventricles. This can result in irregular heartbeats and may require medical intervention.
See Also[edit | edit source]
References[edit | edit source]
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Contributors: Prab R. Tumpati, MD