Caloric deficit

From WikiMD's Wellness Encyclopedia

A caloric deficit is any shortage in the amount of calories consumed relative to the amount of calories required for maintenance of current body weight (energy homeostasis).

A deficit can be created by reducing input/calories consumed (lower food intake, aka dieting).[1] A deficit can also be created by increasing output without a corresponding increase in input. Increased output is created by increasing physical activity,[2] from increased caloric requirements necessary to heal an injury, deficit citation needed (June 2019)

or from growth.[3]  There are also some substances, including Caffeine, which can create a small (3-5%)[4] increase in caloric expenditure, via a variety of pathways that include increasing physical activity levels and increasing Thermogenesis (heat output), and/or by reducing caloric input via appetite suppression.[5][6]  Drugs and herbal treatments creating a more extreme metabolic effect exist; however, they cause extreme increases of heart rate and thermogenesis that can cause death in even very healthy and athletic individuals, and these drugs are not widely sold.[6][7]

As the calories required for energy homeostasis decreases as the organisms's mass decreases, if a moderate deficit is maintained eventually a new (lower) weight will be reached and maintained, and the organism will no longer be at caloric deficit.[2] A permanent severe deficit, on the other hand, which contains too few calories to maintain a healthy weight level, will eventually result in starvation and death. deficit citation needed (June 2019)


To reduce 1 kg of weight, about 7000 kcal deficit is required.[8]

See also[edit | edit source]

References[edit | edit source]

  1. Counting calories: Get back to weight-loss basics Full text, , Mayo Clinic, Accessed on: 12 January 2019.
  2. 2.0 2.1 , Metabolic and Behavioral Compensations in Response to Caloric Restriction: Implications for the Maintenance of Weight Loss, PLOS ONE, Vol. 4 DOI: 10.1371/journal.pone.0004377, PMID: 19198647, PMC: 2634841,
  3. CALORIC NEEDS Full text, www.utmb.edu,
  4. Dulloo, A. G., Normal caffeine consumption: influence on thermogenesis and daily energy expenditure in lean and postobese human volunteers., The American Journal of Clinical Nutrition, Vol. 49(Issue: 1), pp. 44–50, DOI: 10.1093/ajcn/49.1.44, PMID: 2912010, Full text,
  5. Schrader, Patrick, Acute and chronic caffeine administration increases physical activity in sedentary adults, Nutrition Research, 2013, Vol. 33(Issue: 6), pp. 457–463, DOI: 10.1016/j.nutres.2013.04.003,
  6. 6.0 6.1 Dietary Supplements for Weight Loss — Health Professional Fact Sheet Full text, ods.od.nih.gov,
  7. Lu, Yuan-qiang, Clinical features and treatment in patients with acute 2,4-dinitrophenol poisoning, Journal of Zhejiang University. Science. B, Vol. 12(Issue: 3), pp. 189–192, DOI: 10.1631/jzus.B1000265, PMID: 21370503, PMC: 3048933,
  8. Elliott Proctor, Joslin's Diabetes Mellitus: Edited by C. Ronald Kahn ... [et Al.].. online version, Lippincott Williams & Wilkins, ISBN 9780781727969,
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