VG (nerve agent)
VG (nerve agent)
VG is a nerve agent that was originally developed in the United Kingdom in the 1950s. It is a viscous liquid that is colorless and odorless, and it is less volatile than other nerve agents. VG is part of the G-series nerve agents, which also includes GA (Tabun), GB (Sarin), and GD (Soman).
History[edit | edit source]
VG was first synthesized in the 1950s by a team of British scientists. The goal was to create a more potent and longer-lasting nerve agent than those that were currently available. The result was VG, which is also known as Amiton or Tetram in the commercial insecticide industry.
Mechanism of Action[edit | edit source]
Like other nerve agents, VG works by inhibiting the enzyme acetylcholinesterase, which is responsible for breaking down acetylcholine, a neurotransmitter that sends signals in the nervous system. When acetylcholinesterase is inhibited, acetylcholine builds up in the nerve synapses, leading to overstimulation of muscles and glands.
Symptoms and Treatment[edit | edit source]
Exposure to VG can cause a range of symptoms, including convulsions, loss of consciousness, and respiratory failure. Treatment typically involves the administration of atropine and pralidoxime, which help to counteract the effects of the nerve agent.
Use in Warfare[edit | edit source]
While VG has been produced for use in warfare, it is not known to have been used in any conflicts. It is, however, considered a weapon of mass destruction under the Chemical Weapons Convention, and its production, stockpiling, and use is prohibited.
See Also[edit | edit source]
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