Pulmonary-to-systemic shunt
Pulmonary-to-systemic shunt is a medical condition that involves the diversion of blood from the pulmonary circulation to the systemic circulation. This condition is often associated with congenital heart diseases, such as Tetralogy of Fallot and Eisenmenger's syndrome, where the blood bypasses the lungs and does not get oxygenated.
Causes[edit | edit source]
Pulmonary-to-systemic shunt can be caused by several factors, including:
- Congenital heart disease: This is the most common cause of pulmonary-to-systemic shunt. Conditions such as Tetralogy of Fallot, Tricuspid atresia, and Transposition of the great arteries can lead to this condition.
- Pulmonary arteriovenous malformation: This is a rare condition where abnormal connections between the pulmonary arteries and veins lead to shunting of blood.
- Trauma: Injuries to the chest can cause damage to the blood vessels and lead to shunting.
Symptoms[edit | edit source]
The symptoms of pulmonary-to-systemic shunt can vary depending on the severity of the condition. They may include:
- Cyanosis: This is a bluish discoloration of the skin and mucous membranes due to low oxygen levels in the blood.
- Dyspnea: Shortness of breath or difficulty breathing.
- Clubbing: This is a condition where the ends of the fingers and toes become enlarged and rounded.
Diagnosis[edit | edit source]
The diagnosis of pulmonary-to-systemic shunt often involves several tests, including:
- Echocardiogram: This is an ultrasound of the heart that can show the structure and function of the heart.
- Cardiac catheterization: This is a procedure where a catheter is inserted into the heart to measure pressures and oxygen levels.
- Chest X-ray: This can show abnormalities in the heart and lungs.
Treatment[edit | edit source]
The treatment of pulmonary-to-systemic shunt often involves surgery to correct the underlying heart defect. Medications may also be used to manage symptoms.
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