Coxsackievirus-induced cardiomyopathy
| Coxsackievirus-induced cardiomyopathy | |
|---|---|
| Synonyms | Viral myocarditis |
| Pronounce | N/A |
| Specialty | N/A |
| Symptoms | Chest pain, fatigue, shortness of breath, arrhythmia |
| Complications | Heart failure, sudden cardiac death |
| Onset | Sudden or gradual |
| Duration | Variable |
| Types | N/A |
| Causes | Coxsackievirus infection |
| Risks | Immunosuppression, genetic predisposition |
| Diagnosis | Electrocardiogram, echocardiogram, cardiac MRI, endomyocardial biopsy |
| Differential diagnosis | Ischemic heart disease, dilated cardiomyopathy, pericarditis |
| Prevention | Hand hygiene, vaccination (experimental) |
| Treatment | Supportive care, antiviral therapy, immunosuppressive therapy |
| Medication | N/A |
| Prognosis | Variable; can be self-limiting or lead to chronic heart disease |
| Frequency | Rare |
| Deaths | N/A |
- Coxsackieviruses-induced cardiomyopathy are positive-stranded RNA viruses in picornavirus family and the genus enterovirus, acute enterovirus infections such as Coxsackievirus B3 have been identified as the cause of virally induced acute myocarditis, resulting in dilated cardiomyopathy. Dilated cardiomyopathy in humans can be caused by multiple factors including hereditary defects in the cytoskeletal protein dystrophin in Duchenne muscular dystrophy (DMD) patients). A heart that undergoes dilated cardiomyopathy shows unique enlargement of ventricles, and thinning of the ventricular wall that may lead to heart failure. In addition to the genetic defects in dystrophin or other cytoskeletal proteins, a subset of dilated cardiomyopathy is linked to enteroviral infection in the heart, especiall
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