Clinical attachment loss

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Loss of connective tissue attachment in periodontitis


Clinical attachment loss (CAL) refers to the pathological detachment of the periodontal ligament from the tooth surface, resulting in the apical migration of the junctional epithelium and the loss of alveolar bone. It is a critical indicator of periodontal disease progression and is used to assess the severity of periodontitis.

Pathophysiology[edit | edit source]

Clinical attachment loss occurs when the supporting structures of the teeth, including the gingiva, periodontal ligament, and alveolar bone, are destroyed due to inflammatory processes. This destruction is primarily caused by the host's immune response to bacterial plaque accumulation on the teeth. The inflammation leads to the breakdown of collagen fibers in the periodontal ligament and the resorption of alveolar bone, resulting in the detachment of the tooth from its supporting structures.

Measurement[edit | edit source]

The extent of clinical attachment loss is measured using a periodontal probe. The probe is inserted into the gingival sulcus to measure the distance from the cementoenamel junction (CEJ) to the base of the pocket. This measurement is recorded in millimeters and indicates the amount of attachment loss. A higher measurement signifies more severe attachment loss and periodontal disease.

Clinical Significance[edit | edit source]

Clinical attachment loss is a key diagnostic criterion for periodontitis. It helps in classifying the disease into different stages and grades, which are essential for determining the appropriate treatment plan. The presence of clinical attachment loss differentiates periodontitis from gingivitis, which is characterized by inflammation without attachment loss.

Management[edit | edit source]

The management of clinical attachment loss involves controlling the underlying periodontal disease. This includes:

Prevention[edit | edit source]

Preventing clinical attachment loss involves maintaining good oral hygiene to prevent the accumulation of bacterial plaque. This includes:

Related pages[edit | edit source]

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Contributors: Bonnu, Prab R. Tumpati, MD