Warman–Mulliken–Hayward syndrome
Warman–Mulliken–Hayward syndrome is a rare genetic disorder characterized by specific physical abnormalities. The syndrome was first described by Dr. Matthew L. Warman, Dr. Robin T. Mulliken, and Dr. John B. Hayward in the late 20th century.
Symptoms and Signs[edit | edit source]
The primary symptoms of Warman–Mulliken–Hayward syndrome include craniosynostosis, a condition in which the sutures in a baby's skull fuse prematurely, and syndactyly, the fusion of two or more digits. Other symptoms may include micrognathia, a condition characterized by a smaller than normal jaw, and hypertelorism, an abnormally increased distance between two organs or bodily parts, most notably seen in the increased distance between the eyes.
Causes[edit | edit source]
Warman–Mulliken–Hayward syndrome is a genetic disorder, which means it is caused by abnormalities in the individual's genes. The specific gene associated with this syndrome has not yet been identified. It is believed to be inherited in an autosomal dominant manner, which means one copy of the altered gene in each cell is sufficient to cause the disorder.
Diagnosis[edit | edit source]
Diagnosis of Warman–Mulliken–Hayward syndrome is typically based on the presence of characteristic symptoms and signs. Genetic testing may also be used to confirm the diagnosis, although the specific gene associated with this syndrome has not yet been identified.
Treatment[edit | edit source]
Treatment for Warman–Mulliken–Hayward syndrome is symptomatic and supportive. This may include surgery to correct craniosynostosis and syndactyly. Other treatments may be necessary depending on the specific symptoms present in each individual.
See also[edit | edit source]
NIH genetic and rare disease info[edit source]
Warman–Mulliken–Hayward syndrome is a rare disease.
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